We have now succeeded in getting consistently high-quality data from our unique model of the bacterial outer membrane. This involves achieving a highly asymmetric bilayer with phospholipids on the inside and lipopolysaccharide on the outside. To prove the application of this new model we need to show that it behaves like the natural membrane. We know that host primary immune proteins such as lactoferrin and lysozyme bind and disrupt this membrane. We have already demonstrated this for lysozyme. We wish for extra rapid access time to carry out a limited experimental series to show the effect of lactoferrin which has complex and interesting interactions with outer membranes. This will increase the quality of paper and the opportunity to publish in a high impact journal